Monday, 13 September 2010

Subclinical Acidosis

Subclinical acidosis is thought to result from overconsumption of either high-starch concentrates (not applicable to Anky) or an intolerance to pasture grasses rich in fructans (highly likely applicable to Anky). All the info's in the link, but basically it boils down to a lowering of  ph in the hindgut which then becomes overwhelmed with acid and causes serious damage to the intestinal mucosa.
The intestinal lining becomes inflamed and irritated, causing the horse discomfort. The irritation may be severe enough to induce behavior characteristic of colic. Long-term exposure of the intestinal lining to a low-pH environment may negatively affect the absorptive capacities of these structures affecting the ability to absorb nutrients.
Because of the precarious nature of the hindgut of a horse afflicted with subclinical acidosis, it is less able to handle metabolic crises that healthy horses may be able to fend off. Hence, horses with subclinical acidosis are more susceptible to colic and laminitis.
Symptoms include;
  • Decreased feed intake or complete inappetence in severe cases
  • Mild to moderate colic signs of unexplained origin
  • Poor feed conversion efficiency and subsequent weight loss
  • Bloating
  • Loose droppings and behavioural issues
 All these things apply to Anky, but no vet has ever mentioned this to me. Why?
I don't know why, but when I found the article it immediately seemed to me that this might be the missing piece in the jigsaw.  If this is Anky's problem then we can treat it. On verra -we'll see.

4 comments:

Claire said...

no doubt you'll be discussing with your vet!

Di said...

Hehe, yeah, like I've discussed Anky's problem with several vets on several occasions, and nothing has ever been said about this condition!!!!! I don't think so. Luckily I do have more reliable sources of information and advice now!

trudi said...

Wouldn't the ulcer treatment have helped the acidosis or would it not have a delayed enough reaction to reach the hind gut. The cure is bound to be pricey but it could be the answer.

Di said...

"Wouldn't the ulcer treatment have helped the acidosis or would it not have a delayed enough reaction to reach the hind gut."

The gastric ulcer treatment is different (gastrogard - omeprazole)a gastric pump inhibitor, only works in the stomach. You're right though it wouldn't reach the hindgut without losing it's efficacy. The treatment for acidosis is very simple, it's a protected form of sodium bicarbonate, the protection stops it from being destroyed in the stomach so that it can reach the hindgut and lower the Ph, reducing the acidity.
It could be the answer, I hope so, I'm trying not to get my hopes up!!